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Ear & Auditory Pathways

Skull Based Tumors - E Medicine

notes:

Important factors for obtaining ABR:

1. adequate stim strength
2. optimal stim rep rate
3. optimal electrode placement
4. reduction of noise that reaches amplifiers
5. optimal filtering

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Rectangular waves of 100µs duration

 

Electrocochleogram (ECochG )-

Cochelar microphonic-  generated in the receptor cells (hair cells) within the cochlea. Its waveform approximates the sound pressure waveform of the acoustic stimulus (if waveshape of the acoustic stimulus is reversed in polarity, the cochlear microphonic is also inverted).

8th CN action potential- generated by depolarization within the distal (cochlear) ends of the auditory nerve axons, which have been activated by excitatory synaptic input from the cochlear hair cells. It is recorded as a phasic negativity in the middle ear or extratympanic recording site irrespective of the stimulus polarity.  Multiple volleys are typically illicited (N1 and N2, sometimes N3 components).

 

Auditory Evoked Potentials:

Short Latency- under 10 ms. BAEPs

Middle Latency- 10-50 ms. Most likely generatedwithin the cerebral cortex, including the primary auditory cortex. Affected by anesthesia, so not useful for IOM, but they have been used to assess the depth of anesthesia.

Long Latency- over 50 ms.  Predominantly generated in the cortex including cortical association areas. Suppressed by anesthesia- not useful in IOM.

BAEPs

BAEPs can be used to monitor acoustically evoked activity within the auditory pathways up through the level of the mesencephalon; they are not useful for monitoring the pathways rostral to this. They are most often used to monitor surgery for eighth nerve tumors such as vestibular schwannomas (formerly called acoustic neuromas) and for tumors or vascular abnormalities within the posterior fossa, both extra-axial and within the substance of the brainstem. BAEP monitoring can help to avoid excessive eighth nerve stretch from cerebellar retraction, which can cause hearing loss, during surgery in the cerebellopontine angle.

 

BAEP components are composites of contributions of multiple generators. The complexity of the generators of human BAEPs derives in part from the pattern of connections within the auditory pathways, with ascending fibers both synapsing in and bypassing various relay nuclei.

It also reflects the presence of at least two bursts of activity in the auditory nerve (corresponding to the N1 and N2 components of the eighth nerve compound action potentials in the ECochG), which can drive the more rostral pathways.Several different structures within the infratentorial auditory pathways may be active and generating field potentials simultaneously.

 

Wave I

Arises from the 1st volley of action potentials in the auditory nerve in the most distal portion of the nerve. Represents the N1 component of the 8th CN compound action potential in the ECochG. Because wave I arises from the most distal portion of the auditory nerve, it may persist after the nerve is sectioned at a more proximal location, such as during surgery for eighth nerve tumors.

Wave II

Originates, in part, in neural activity that began as the N1 component of the eighth nerve compound action potential and has propagated from the distal auditory nerve to its proximal end and to the cochlear nucleus. However, the activity at this point in the auditory pathway occurs simultaneously with the second auditory nerve volley, the N2 component of the eighth nerve compound action potential, in the distal nerve (Gersdorff, 1982). The latter contributes to the scalp-recorded BAEP in the same manner as the N1 component did when it was at the same location. This can cause persistence of a wave II in cases where the proximal eighth nerve has been destroyed (Legatt, 2005). With regard to the more proximal generator of wave II, the relative contribution of activity in auditory nerve fibers within the proximal nerve and of activity in cochlear nucleus neurons has been a subject of controversy (Legatt et al., 1988). This proximal generator is the major determinant of the scalp topography of this BAEP component over the dorsal part of the head

Wave III

Predominantly originates in the caudal pontine tegmentum, including the region of the superior olivary complex, though a contribution from continued activity at the level of the cochlear nucleus cannot be ruled out (Legatt, 2005). Ascending projections from the cochlear nucleus are bilateral, so wave III may receive contributions from brainstem auditory structures both ipsilateral and contralateral to the stimulated ear. In patients with asymmetrical lesions of the brainstem, wave III abnormalities are usually most pronounced following stimulation of the ear ipsilateral to the lesion (Brown et al., 1981; Oh et al., 1981; Faught and Oh, 1985), though occasionally they aremore pronounced following contralateral stimulation (Stockard and Rossiter, 1977).

Waves IV and V

Waves IV and Vare often fused into a IV–V complex, and their anatomical generators are most likely in close anatomical proximity or overlapping, since they are usually either both affected or both unaffected by brainstem lesions. They may, however, be differentially affected by intraoperative brainstem damage.

Wave IV appears to reflect activity predominantly in ascending auditory fibers within the dorsal and rostral pons, just caudal to the inferior colliculus

Wave V predominantly reflects activity at the level of the inferior colliculus, perhaps including activity in the rostral portion of the lateral lemniscus as it terminates in the inferior colliculus.

As is the case with wave III, wave V abnormalities due to unilateral brain stemlesions are usually most pronounced following stimulation of the ear ipsilateral to the lesion , though there are exceptions

Waves VI

Waves VI and VII are absent in some normal subjects. While they may in part reflect activity in more rostral structures such as the medial geniculate nucleus, they also receive contributions from activity in the inferior colliculus; the latter generator may cause persistence of these waves in patients with auditory pathway damage rostral to the inferior colliculus. Therefore, BAEPs cannot be used to assess or monitor the auditory pathways rostral to the mesencephalon.

STIMULATION

 

Recording